TUESDAY, 18 MAY 2021 at 1500 BST

A material’s capacity for limited deformation is a critical aspect of toughness as this enables the local dissipation of stresses that would otherwise cause fracture. Such inelastic deformation mechanisms are diverse; they include dislocation motion in crystalline materials, in-situ phase-transformations in certain metals and ceramics, sliding of collagen fibrils in bone, the rotation of fibrils in skin, frictional motion between mineral “platelets” in seashells, and through mechanisms that also cause fracture such as shear-banding in glasses and microcracking in rocks.

Resistance to fracture is thus a compromise: either a combination of the mutually exclusive properties of strength and deformability, as in intrinsic toughness, or between intrinsic and extrinsic (shielding) mechanisms that act to induce toughness, respectively, ahead or behind, the tip.  We examine the interplay between such mechanisms in biological materials, including skin and bone, seashells and fish scales, in high-temperature materials, such as ceramic-matrix composites and nuclear graphite, and in multiple-element metallic alloys, namely bulk-metallic glasses and high-entropy alloys, in order to seek the many diverse processes that are the source of the fracture resistance of materials.

 

Register to attend 'On the Fracture Toughness of Biological and Engineering Structural Materials' at 1500GMT on Tuesday, 18 May 2021.